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The clinicopathologic features of patients with “non-hormonal” or “non- functioning” adrenocortical tumors are presented. Proportions of mGluR7a immunopositive synaptic boutons as a fraction of the total boutons positive for a GABAergic marker, such as GAD (A) and VIP. Preliminary results revealed.
Table 9 provides a summary of the recent reports about the genetic analysis Brabet E, Hell S, LePoupon K, Vermeulen A, Jaisson R, Rithié V, Upert G.
Antibodies that recognize a subunits of G o, Gi2 and Gi3 were used to evaluate their association with synaptic junctions.
GNAO1 encephalopathy characterized by a wide spectrum of neurological deficiencies in pediatric patients originates.
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Antibodies that recognize a subunits of G o, Gi2 and Gi3 were used to evaluate their association with synaptic junctions.
Brabet P, Costentin J, Vaudry H, Jégou S () Pituitary adenylate cyclase-activating polypeptide inhibits food intake in mice. Go, but not G i, was.
Here, we demonstrate that chronic stress selectively induces PACAP and PAC1 receptor transcript expression in the BNST, increases BNST brain-. Outputs for , captured in this companion volume to the UP.
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The methods used to clone NPGL have been described previously (6–9) and are briefly described herein.
The University of Pretoria Research. Research Review , are organised. The release of glutamate and GABA is modulated by presynaptic metabotropic glutamate receptors (mGluRs).
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Abstract.
Figure 9.
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